Stroke-Induced, Moderate, Acquired Apraxia of Speech and Nonfluent Aphasia
Apraxia of speech (AOS) is an acquired neurogenic communication disorder resulting from disrupted planning/programming of motor speech production. Although, historically, there has been some disagreement regarding the characteristic features of AOS, most experts in the field agree that the following features are characteristic of AOS: sound errors (i.e., distortions, often perceived as sound substitutions), increased segment and/or intersegment durations, and slowed speech rate with a tendency to segregate syllables and equalize stress across syllables.1 , 2 Other common features that may be present, but are not unique to AOS include articulatory groping (silent or audible), increased errors with increased length and/or complexity of utterances, motor perseveration, difficulty initiating speech, self-awareness of errors, improved automatic speech production, and periods of errorless speech (e.g., social conventions). Differentiating AOS from other neurogenic speech and language disorders (e.g., aphasia with phonemic paraphasia) has also been an area of concern for researchers and clinicians alike. This case will present the history and assessment tasks necessary to adequately differentiate and diagnose AOS with subsequent intervention as indicated.